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Info from the vet.....
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Kelvin first<br>
BUN was very elevated +100, normal is 25, normal for CRF with
treatment is 60 - 80, but at first diagnosis it's usually 150 or more.
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We aren't up that far and the vet wants a retest when we do Gus's
T4 to see if it was just dehydration since all his veins blew or failed
when they
were trying to pull blood.
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This was not unexpected due to the ehrlickia diagnosis in December
it just means he's further along in the disease that we assumed. I will
be starting a CRF diet for him anyway after the next test if it is
still elevated.
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One more reason not to EVER adopt him out.<br>
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Now Gus<br>
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valign="top" width="100%">We have results back already Vet is using
the lab in Austin now so it's WAY faster.
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Follicular Nevus
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This is what I have found on it so far:
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<span style="text-decoration: underline;">Nevi: An hamartoma is a
malformation with tumoral aspect, formed by components which are normal
parts of the organ in which it develops but which are arranged
erroneously (CIVATTE). A nevus is a cutaneous hamartoma which may arise
from any skin component. Nevi are rare, congenital or not and the
mechanism of their formation is unknown.</span>
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a. Collagenous nevi are single or multiple nodules (0.5 to a few
cm) with sometimes an "orange peel" appearance. Large nodular areas of
collagene hyperplasia characterize the disease histopathologically. In
German Shepherds multiple collagenous nevi may appear, particularly on
the limbs (leading to lameness), associated with renal adenocarcinomas
and uterine leiomyomas. This syndrome, due to an autosomal dominant
gene, is called "nodular dermatofibrosis".
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b. Organoid or pilo-sebaceous nevi are solitary or multiple
lesions, pedunculated or linear, seen on the hand or the limbs.
Hyperplasia of hair follicles and sebaceous glands are the main
histopathological features (apocrine sweat glands remaining normal).
Focal adnexal dysplasia is similar but there is inflammation and a
bizarre configuration.
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c. Vascular nevi or angioma are often multiple lesions seen in the
scrotum but also all over the body surface. Bleeding is frequent.
Histopathology shows a cavernous telangiectasia.
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d. Sebaceous nevi are very rare congenital lesions characterized by
sebaceous hyperplasia involving multiple folliculosebaceous units and
papillomatosis. Miniature Schnauzer comedo syndrome can be considered
as a pseudoneoplasm.
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e. Epidermal nevi are hyperpigmented linear plaques seen on the
trunk or limbs, histopathologically characterized by orthokeratotic
hyperkeratosis and papillomatosis.
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<b>f. Follicular nevi are plaques (several cms in diameter)
containing clusters of normal anagen primary hair follicles that are
large and extend deeply.</b>
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<span style="text-decoration: underline;">g. Treatment: nevi
should be surgically excised when feasible. </span>
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Theory is that Gus's nevi form into pilomatricoma if left alone.
they will all come out from now on. Lab wants to use him as a study
since it's not remotely normal in bassets.<br>
<br>
Carmen<br>
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